Thermal or chemical burns lead to the destruction HaloHair Gummies Review of the skin and blood vessels. The blood capillaries at the site become dilated and more permeable causing loss of fluid, electrolytes and plasma proteins into the tissues. This causes oedema and formation of blisters. Fluid loss causes dehydration and hypovolaemic shock. The blood vessels around the burn become thrombosed resulting in a decrease in circulation to the area thereby aggravating the tissue damage.
Sodium is lost into the oedema fluid. Further loss of blood proteins into the fluid promotes oedema, causing decrease in intravascular colloidal osmotic pressure. Potassium is lost from the damaged cells leading to temporary hypokalaemia. More of the potassium is excreted by the kidneys.
Hypoxia develops as a result of the hypovolaemia, thrombosed blood vessels and sluggish circulation which cause decrease tissue perfusion. The ensuing hypoxia leads to metabolic acidosis. Fluid and sodium loss cause an increase in the production of aldosterone and the anti-diuretic hormone (ADH). The aldosterone causes the kidneys to conserve sodium, while the ADH brings about conservation of water resulting in the passage of scanty, concentrated urine (oliguria).
Injury to the skin and nerve endings produces severe pain and loss of function of the skin. Pain is severe in partial thickness burns, but in full thickness burns there is loss of sensation. The open wound created ensures an easy access for micro-organisms which infect the wound. With infection, there is the release of toxins which cause pyrexia, malaise and delayed wound healing. Absorption of decomposed products from dead tissues leads to toxaemia.
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